Best Selling Books by Paul Ryan

This is the story of what happens to a boy on the first day of a revolution. The boy is named Jack. He is 17. He is your guide on one hell of a day.

Killing the Rising Sun Read Killing the Rising Sun on your PC, Mac, smart phone, table, ipad or Kindle device. Upon hearing that the United States had entered the war, British Prime Minister Winston Churchill had said "we have won the war." Indeed, America''s machine of war began to rev up as they started to get involved in two theaters of war. The first theater was the European theater, where their involvement in fighting Germany was relatively minimal at best. The truth is, while many Americans like to look at Germany as being the main enemy of World War II, to America, the Japanese were more of the enemy than the Germans. While America gave significant support, military assistance and weapons to their allies who were fighting against the German front, the USA''s primary goal was to fight against the Japanese. Thus, sparked the greatest military campaign America had ever seen before, thus began the Pacific War.The purpose of this book is to give you an overview of the American campaign against Japan and to show how they were able to defeat the Japanese despite the fanatical strength of their opponents. First, we are going to look at the reason why the Japanese attacked America, how America responded, and look at the ultimate moment of victory for America, the dropping of the atomic bomb. If you''re someone who''s curious about learning about why Japan chose to wage war against us, how the war went down and what made America so strong against the Japanese, then read on! Here Is A Preview Of What You''ll Learn... Fanatical Japan The battle of Midway The battle of Iwo Jima The horrors of Okinawa The nuclear age Much, much more! Download your copy today!

We studied the light-induced reactions of the (6-4) photolyase, a flavoenzyme of the cryptochrome/photolyase family that repairs the UV-induced (6-4) photodamage in DNA with the aid of blue light. We studied this photorepair reaction as well as the light-induced cofactor reduction called photoactivation that the enzyme uses to bring itself to a repair-active state in the (6-4) photolyase from Xenopus laevis. We have studied the photoactivation of the FADox cofactor of the enzyme using femtosecond polarised transient absorption spectroscopy. We observed a sub-picosecond electron transfer (~400 fs) after excitation of the FADox cofactor. We were able to characterise a tryptophan residue as the electron donor. We sought to differentiate the spectroscopically identical but differently oriented tryptophan residues within the protein''s photoactivation site by transient anisotropy measurements. Our results suggest that the photoactivation mechanism is not fully compatible with the mechanism thought to be conserved among photolyases: an electron transfer mechanism via electron hopping along a chain of three highly conserved tryptophan residues.Using series of single turnover flashes, we have found that the repair reaction proceeds by a successive two-photon mechanism. The first photon converts the (6-4) lesion into a metastable intermediate X, the lifetime of which is ~2 min. Absorption of a second photon within the lifetime of X results to the restoration of intact nucleobases. In light of our findings, the reaction was also studied by femtosecond transient absorption spectroscopy.

In experiments reported here, visual and vestibular stimuli were manipulated independently. Observers made judgments about body orientation or self-motion. Results show that visual and vestibular senses are highly complimentary, in that they provide very similar types of information. Results also show that self-motion stimuli influence body orientation perception, and body orientation stimuli influence self-motion perception.

The effects of graphite surface modification on fatigue crack growth resistance of the graphite/epoxy interface were examined using flexural peel specimens. Edge surfaces of pryolitic graphite were treated in an oxygen plasma for various lengths of time and subsequently bonded to a toughened epoxy to form flexural peel specimens. Fatigue crack growth rates were measured for the plasma treated and untreated specimens as a function of strain energy release rate. Fatigue crack growth resistance of plasma treated specimens was notably higher than that of untreated specimens, with the fatigue threshold doubled at an optimal treatment time. X-ray photoelectron spectroscopy, electron microscopy, and surface profilometry studies indicated that both surface chemistry and surface morphology of the graphite were changed by the plasma treatment. High temperature annealing was used to restore the original surface chemistry while retaining the etched surface morphology. Fatigue experiments were then performed on the heat treated specimens to separate the chemical and morphological effects on resistance to crack growth. Chemical modification turned out to be a secondary effect, contributing less than 13% to the overall fatigue crack growth resistance. A micro-mechanical model is proposed which explains the relative effects of surface chemistry and morphology on fatigue crack growth resistance along the graphite/epoxy interface.