Most Popular Books by David Rhodes

This work has been selected by scholars as being culturally important, and is part of the knowledge base of civilization as we know it. This work is in the "public domain in the United States of America, and possibly other nations. Within the United States, you may freely copy and distribute this work, as no entity (individual or corporate) has a copyright on the body of the work. Scholars believe, and we concur, that this work is important enough to be preserved, reproduced, and made generally available to the public. We appreciate your support of the preservation process, and thank you for being an important part of keeping this knowledge alive and relevant.

Do you love roller coasters? Then maybe you could get a job designing one. There are more jobs for those who love an adrenaline rush than you probably ever realized. From adventure guides to bomb squad technicians, readers will learn about various STEM and STEAM jobs for thrill seekers and what it takes to get one.

This is a collection of daily meditations for Lent, Holy Week and Easter. The author draws on the social justice aspect of Christ''s teaching, relating the gospel to everyday life. Each Sunday''s meditation is based on a phrase from the Lord''s Prayer; this phrase is echoed in the following week''s meditations. It should be useful to anyone seeking to live out the love of God in the world.

Do you love playing video games? Did you know you can get a job helping to create one? There are more jobs for those who love gaming than you probably ever realized. From animators to script writers, readers will learn about various STEM and STEAM gaming jobs and what it takes to get one.

The first book in the Following God for Young Adults series, God Is...is an eight-week study designed to explore the many sides of the character of God so that young adults (teens and twenty-somethings) will begin to understand God better and allow the truth about Him to lead to growth in their lives.

The third book in the Following God for Young Adults series, Redefining Normal turns us upside down as we examine the kind of people God wants us to become. This eight-week study challenges the norm of our culture and Christianity and urges young adults to live under grace and grow in holiness. The challenge presented is not only to live within God''s moral boundaries but also to become people of greater faith, hope, and love.

A time traveling madman believes humanity has lost its way. To force society to follow his demands for change, he unleashes dinosaurs on a horrified world. The dinosaurs, which he considers justice, will disappear only after every country follows his rules. A team sent to capture him will have to survive lies, deceit, dinosaurs, and history as they attempt to save the world...and each other.

Walsh''s pioneering catalogue of the Harvard collection of 15th-century printed books was published in five volumes. The First Supplement describes 202 new incunabula at Harvard: 67 complete or nearly complete copies and 135 single leaves or fragments, representing a total of 173 editions, including 110 not in Walsh''s original five volumes.

Do you love watching racing cars? Then maybe you can get a job making them go faster. There are more jobs for gearheads than you probably ever realized. From classic car restorer to motorcycle designer, readers will learn about various STEM and STEAM jobs for gearheads and what it takes to get one.

This work has been selected by scholars as being culturally important, and is part of the knowledge base of civilization as we know it. This work was reproduced from the original artifact, and remains as true to the original work as possible. Therefore, you will see the original copyright references, library stamps (as most of these works have been housed in our most important libraries around the world), and other notations in the work. This work is in the public domain in the United States of America, and possibly other nations. Within the United States, you may freely copy and distribute this work, as no entity (individual or corporate) has a copyright on the body of the work.As a reproduction of a historical artifact, this work may contain missing or blurred pages, poor pictures, errant marks, etc. Scholars believe, and we concur, that this work is important enough to be preserved, reproduced, and made generally available to the public. We appreciate your support of the preservation process, and thank you for being an important part of keeping this knowledge alive and relevant.

Indulge your senses in the grimy and perverse fetish underworld of Los Angeles. Mystic exotic dancers; stylish dominatrixes; burnt-out detectives are but a few of the psychologically tormented characters in this noir novel. The characters unravel their lives while around them a serial killer lurks. To stimulate the reading experience this erotic thriller also contains sensuous visuals.

Skeletal manifestations including short stature, osteoporosis, kyphoscoliosis, and tibial dysplasia cumulatively affect approximately 70% of patients with neurofibromatosis type 1 (NF1). Tibial pseudarthrosis, the chronic non-union of a spontaneous fracture, is a debilitating skeletal malady affecting young children with NF1. These non-healing fractures respond poorly to treatment and often require amputation of the affected limb due to limited understanding of the causative mechanisms. To better understand the cellular and molecular pathogenesis of these osseous defects, we have established a new mouse model which recapitulates a spectrum of skeletal pathologies frequently observed in patients with NF1. Nf1flox/-;Col2.3Cre mice, harboring Nf1 nullizygous osteoblasts on a Nf1+/- background, exhibit multiple osseous defects which are closely reminiscent of those found in NF1 patients, including runting (short stature), bone mass deficits, spinal deformities, and tibial fracture non-union. Through adoptive bone marrow transfer studies, we have demonstrated that the Nf1 haploinsufficient hematopoietic system pivotally mediates the pathogenesis of bone loss and fracture non-union in Nf1flox/-;Col2.3Cre mice. By genetic ablation of a single Nf1 allele in early myeloid development, under the control of LysMCre, we have further delineated that Nf1 haploinsufficient myeloid progenitors and osteoclasts are the culprit lineages mediating accelerated bone loss. Interestingly, conditional Nf1 haploinsufficiency in mature osteoclasts, induced by CtskCre, was insufficient to trigger enhanced lytic activity. These data provide direct genetic evidence for Nf1''s temporal significance as a gatekeeper of the osteoclast progenitor pool in primitive myelopoiesis. On the molecular level, we found that transforming growth factor-beta1 (TGF-?1), a primary mediator in the spatiotemporal coupling of bone remodeling, is pathologically overexpressed by five- to six- fold in both NF1 patients and in mice. Nf1 deficient osteoblasts, the principal source of TGF-?1 in the bone matrix, overexpress TGF-?1 in a gene dosage dependent fashion. Moreover, p21Ras dependent hyperactivation of the Smad pathway accentuates responses to pathological TGF-?1 signals in Nf1 deficient bone cells. As a proof of concept, we demonstrate that pharmacologic T?RI kinase inhibition can rescue bone mass defects and prevent tibial fracture non-union in Nf1flox/-;Col2.3Cre mice, suggesting that targeting TGF-?1 signaling in myeloid lineages may provide therapeutic benefit for treating NF1 skeletal defects.

Oak Junction, Iowa, a small town where nothing ever happens, is changed forever when a mysterious man with an old and powerful book appears there.

An analysis of plasma polymers produced from non-traditional monomers.